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Showing 1 to 12 of 109 entries
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Anti-Vascular endothelial growth factor therapy impairs endothelial function of retinal microcirculation in colon cancer patients - an observational study.

Experimental & translational stroke medicine

Reimann M, Folprecht G, Haase R, Trautmann K, Ehninger G, Reichmann H, Ziemssen F, Ziemssen T.
PMID: 23668549
Exp Transl Stroke Med. 2013 May 13;5(1):7. doi: 10.1186/2040-7378-5-7.

BACKGROUND: To assess acute effects of bevacizumab (anti-VEGF therapy) on cerebral microvessels and systemic cardiovascular regulation.DESIGN AND SUBJECTS: 20 consecutive patients with colorectal cancer (median age: 60.4 years, range 45.5-73.9 years) received bevacizumab intravenously (5 mg/kg) uncoupled of chemotherapy....

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Reimann M, Folprecht G, Haase R, et al. Anti-Vascular endothelial growth factor therapy impairs endothelial function of retinal microcirculation in colon cancer patients - an observational study. Exp Transl Stroke Med. 2013;5(1):7doi: 10.1186/2040-7378-5-7.
Reimann, M., Folprecht, G., Haase, R., Trautmann, K., Ehninger, G., Reichmann, H., Ziemssen, F., & Ziemssen, T. (2013). Anti-Vascular endothelial growth factor therapy impairs endothelial function of retinal microcirculation in colon cancer patients - an observational study. Experimental & translational stroke medicine, 5(1), 7. https://doi.org/10.1186/2040-7378-5-7
Reimann, Manja, et al. "Anti-Vascular endothelial growth factor therapy impairs endothelial function of retinal microcirculation in colon cancer patients - an observational study." Experimental & translational stroke medicine vol. 5,1 (2013): 7. doi: https://doi.org/10.1186/2040-7378-5-7
Reimann M, Folprecht G, Haase R, Trautmann K, Ehninger G, Reichmann H, Ziemssen F, Ziemssen T. Anti-Vascular endothelial growth factor therapy impairs endothelial function of retinal microcirculation in colon cancer patients - an observational study. Exp Transl Stroke Med. 2013 May 13;5(1):7. doi: 10.1186/2040-7378-5-7. PMID: 23668549; PMCID: PMC3658870.
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Outcome of experimental stroke in C57Bl/6 and Sv/129 mice assessed by multimodal ultra-high field MRI.

Experimental & translational stroke medicine

Pham M, Helluy X, Braeuninger S, Jakob P, Stoll G, Kleinschnitz C, Bendszus M.
PMID: 20230638
Exp Transl Stroke Med. 2010 Mar 15;2:6. doi: 10.1186/2040-7378-2-6.

Transgenic mice bred on C57Bl/6 or Sv/129 genetic background are frequently used in stroke research. It is well established that variations in cerebrovascular anatomy and hemodynamics can influence stroke outcome in different inbred mouse lines. We compared stroke development...

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Pham M, Helluy X, Braeuninger S, et al. Outcome of experimental stroke in C57Bl/6 and Sv/129 mice assessed by multimodal ultra-high field MRI. Exp Transl Stroke Med. 2010;2:6doi: 10.1186/2040-7378-2-6.
Pham, M., Helluy, X., Braeuninger, S., Jakob, P., Stoll, G., Kleinschnitz, C., & Bendszus, M. (2010). Outcome of experimental stroke in C57Bl/6 and Sv/129 mice assessed by multimodal ultra-high field MRI. Experimental & translational stroke medicine, 26. https://doi.org/10.1186/2040-7378-2-6
Pham, Mirko, et al. "Outcome of experimental stroke in C57Bl/6 and Sv/129 mice assessed by multimodal ultra-high field MRI." Experimental & translational stroke medicine vol. 2 (2010): 6. doi: https://doi.org/10.1186/2040-7378-2-6
Pham M, Helluy X, Braeuninger S, Jakob P, Stoll G, Kleinschnitz C, Bendszus M. Outcome of experimental stroke in C57Bl/6 and Sv/129 mice assessed by multimodal ultra-high field MRI. Exp Transl Stroke Med. 2010 Mar 15;2:6. doi: 10.1186/2040-7378-2-6. PMID: 20230638; PMCID: PMC2847985.
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Induction of neuro-protective/regenerative genes in stem cells infiltrating post-ischemic brain tissue.

Experimental & translational stroke medicine

Yilmaz G, Alexander JS, Erkuran Yilmaz C, Granger DN.
PMID: 20509949
Exp Transl Stroke Med. 2010 May 28;2(1):11. doi: 10.1186/2040-7378-2-11.

BACKGROUND: Although the therapeutic potential of bone marrow-derived stromal stem cells (BMSC) has been demonstrated in different experimental models of ischemic stroke, it remains unclear how stem cells (SC) induce neuroprotection following stroke. In this study, we describe a...

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Yilmaz G, Alexander JS, Erkuran Yilmaz C, et al. Induction of neuro-protective/regenerative genes in stem cells infiltrating post-ischemic brain tissue. Exp Transl Stroke Med. 2010;2(1):11doi: 10.1186/2040-7378-2-11.
Yilmaz, G., Alexander, J. S., Erkuran Yilmaz, C., & Granger, D. N. (2010). Induction of neuro-protective/regenerative genes in stem cells infiltrating post-ischemic brain tissue. Experimental & translational stroke medicine, 2(1), 11. https://doi.org/10.1186/2040-7378-2-11
Yilmaz, Gokhan, et al. "Induction of neuro-protective/regenerative genes in stem cells infiltrating post-ischemic brain tissue." Experimental & translational stroke medicine vol. 2,1 (2010): 11. doi: https://doi.org/10.1186/2040-7378-2-11
Yilmaz G, Alexander JS, Erkuran Yilmaz C, Granger DN. Induction of neuro-protective/regenerative genes in stem cells infiltrating post-ischemic brain tissue. Exp Transl Stroke Med. 2010 May 28;2(1):11. doi: 10.1186/2040-7378-2-11. PMID: 20509949; PMCID: PMC2893124.
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Neuronal precursor cell proliferation in the hippocampus after transient cerebral ischemia: a comparative study of two rat strains using stereological tools.

Experimental & translational stroke medicine

Kelsen J, Larsen MH, Sørensen JC, Møller A, Frøkiaer J, Nielsen S, Nyengaard JR, Mikkelsen JD, Rønn LC.
PMID: 20370927
Exp Transl Stroke Med. 2010 Apr 06;2:8. doi: 10.1186/2040-7378-2-8.

BACKGROUND: We are currently investigating microglial activation and neuronal precursor cell (NPC) proliferation after transient middle cerebral artery occlusion (tMCAo) in rats. This study aimed: (1) to investigate differences in hippocampal NPC proliferation in outbred male spontaneously hypertensive rats...

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Kelsen J, Larsen MH, Sørensen JC, et al. Neuronal precursor cell proliferation in the hippocampus after transient cerebral ischemia: a comparative study of two rat strains using stereological tools. Exp Transl Stroke Med. 2010;2:8doi: 10.1186/2040-7378-2-8.
Kelsen, J., Larsen, M. H., Sørensen, J. C., Møller, A., Frøkiaer, J., Nielsen, S., Nyengaard, J. R., Mikkelsen, J. D., & Rønn, L. C. (2010). Neuronal precursor cell proliferation in the hippocampus after transient cerebral ischemia: a comparative study of two rat strains using stereological tools. Experimental & translational stroke medicine, 28. https://doi.org/10.1186/2040-7378-2-8
Kelsen, Jesper, et al. "Neuronal precursor cell proliferation in the hippocampus after transient cerebral ischemia: a comparative study of two rat strains using stereological tools." Experimental & translational stroke medicine vol. 2 (2010): 8. doi: https://doi.org/10.1186/2040-7378-2-8
Kelsen J, Larsen MH, Sørensen JC, Møller A, Frøkiaer J, Nielsen S, Nyengaard JR, Mikkelsen JD, Rønn LC. Neuronal precursor cell proliferation in the hippocampus after transient cerebral ischemia: a comparative study of two rat strains using stereological tools. Exp Transl Stroke Med. 2010 Apr 06;2:8. doi: 10.1186/2040-7378-2-8. PMID: 20370927; PMCID: PMC2868803.
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On the importance of long-term functional assessment after stroke to improve translation from bench to bedside.

Experimental & translational stroke medicine

Freret T, Schumann-Bard P, Boulouard M, Bouet V.
PMID: 21682914
Exp Transl Stroke Med. 2011 Jun 18;3:6. doi: 10.1186/2040-7378-3-6.

Despite extensive research efforts in the field of cerebral ischemia, numerous disappointments came from the translational step. Even if experimental studies showed a large number of promising drugs, most of them failed to be efficient in clinical trials. Based...

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Freret T, Schumann-Bard P, Boulouard M, et al. On the importance of long-term functional assessment after stroke to improve translation from bench to bedside. Exp Transl Stroke Med. 2011;3:6doi: 10.1186/2040-7378-3-6.
Freret, T., Schumann-Bard, P., Boulouard, M., & Bouet, V. (2011). On the importance of long-term functional assessment after stroke to improve translation from bench to bedside. Experimental & translational stroke medicine, 36. https://doi.org/10.1186/2040-7378-3-6
Freret, Thomas, et al. "On the importance of long-term functional assessment after stroke to improve translation from bench to bedside." Experimental & translational stroke medicine vol. 3 (2011): 6. doi: https://doi.org/10.1186/2040-7378-3-6
Freret T, Schumann-Bard P, Boulouard M, Bouet V. On the importance of long-term functional assessment after stroke to improve translation from bench to bedside. Exp Transl Stroke Med. 2011 Jun 18;3:6. doi: 10.1186/2040-7378-3-6. PMID: 21682914; PMCID: PMC3141537.
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Experimental models for analysis of oligodendrocyte pathophysiology in stroke.

Experimental & translational stroke medicine

Arai K, Lo EH.
PMID: 20150984
Exp Transl Stroke Med. 2009 Oct 24;1:6. doi: 10.1186/2040-7378-1-6.

White matter damage is a clinically important part of stroke. However, compared to the mechanisms of neuronal injury in gray matter, white matter pathophysiology remains relatively understudied and poorly understood. This mini-review aims at summarizing current knowledge on experimental...

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Arai K, Lo EH. Experimental models for analysis of oligodendrocyte pathophysiology in stroke. Exp Transl Stroke Med. 2009;1:6doi: 10.1186/2040-7378-1-6.
Arai, K., & Lo, E. H. (2009). Experimental models for analysis of oligodendrocyte pathophysiology in stroke. Experimental & translational stroke medicine, 16. https://doi.org/10.1186/2040-7378-1-6
Arai, Ken, and Lo, Eng H. "Experimental models for analysis of oligodendrocyte pathophysiology in stroke." Experimental & translational stroke medicine vol. 1 (2009): 6. doi: https://doi.org/10.1186/2040-7378-1-6
Arai K, Lo EH. Experimental models for analysis of oligodendrocyte pathophysiology in stroke. Exp Transl Stroke Med. 2009 Oct 24;1:6. doi: 10.1186/2040-7378-1-6. PMID: 20150984; PMCID: PMC2820444.
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Rodent models of focal cerebral ischemia: procedural pitfalls and translational problems.

Experimental & translational stroke medicine

Braeuninger S, Kleinschnitz C.
PMID: 20150986
Exp Transl Stroke Med. 2009 Nov 25;1:8. doi: 10.1186/2040-7378-1-8.

Rodent models of focal cerebral ischemia are essential tools in experimental stroke research. They have added tremendously to our understanding of injury mechanisms in stroke and have helped to identify potential therapeutic targets. A plethora of substances, however, in...

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Braeuninger S, Kleinschnitz C. Rodent models of focal cerebral ischemia: procedural pitfalls and translational problems. Exp Transl Stroke Med. 2009;1:8doi: 10.1186/2040-7378-1-8.
Braeuninger, S., & Kleinschnitz, C. (2009). Rodent models of focal cerebral ischemia: procedural pitfalls and translational problems. Experimental & translational stroke medicine, 18. https://doi.org/10.1186/2040-7378-1-8
Braeuninger, Stefan, and Kleinschnitz, Christoph. "Rodent models of focal cerebral ischemia: procedural pitfalls and translational problems." Experimental & translational stroke medicine vol. 1 (2009): 8. doi: https://doi.org/10.1186/2040-7378-1-8
Braeuninger S, Kleinschnitz C. Rodent models of focal cerebral ischemia: procedural pitfalls and translational problems. Exp Transl Stroke Med. 2009 Nov 25;1:8. doi: 10.1186/2040-7378-1-8. PMID: 20150986; PMCID: PMC2820446.
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Dynamics of neuroinflammation in the macrosphere model of arterio-arterial embolic focal ischemia: an approximation to human stroke patterns.

Experimental & translational stroke medicine

Walberer M, Rueger MA, Simard ML, Emig B, Jander S, Fink GR, Schroeter M.
PMID: 21171972
Exp Transl Stroke Med. 2010 Dec 20;2(1):22. doi: 10.1186/2040-7378-2-22.

BACKGROUND: Neuroinflammation evolves as a multi-facetted response to focal cerebral ischemia. It involves activation of resident glia cell populations, recruitment of blood-derived leucocytes as well as humoral responses. Among these processes, phagocyte accumulation has been suggested to be a...

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Walberer M, Rueger MA, Simard ML, et al. Dynamics of neuroinflammation in the macrosphere model of arterio-arterial embolic focal ischemia: an approximation to human stroke patterns. Exp Transl Stroke Med. 2010;2(1):22doi: 10.1186/2040-7378-2-22.
Walberer, M., Rueger, M. A., Simard, M. L., Emig, B., Jander, S., Fink, G. R., & Schroeter, M. (2010). Dynamics of neuroinflammation in the macrosphere model of arterio-arterial embolic focal ischemia: an approximation to human stroke patterns. Experimental & translational stroke medicine, 2(1), 22. https://doi.org/10.1186/2040-7378-2-22
Walberer, Maureen, et al. "Dynamics of neuroinflammation in the macrosphere model of arterio-arterial embolic focal ischemia: an approximation to human stroke patterns." Experimental & translational stroke medicine vol. 2,1 (2010): 22. doi: https://doi.org/10.1186/2040-7378-2-22
Walberer M, Rueger MA, Simard ML, Emig B, Jander S, Fink GR, Schroeter M. Dynamics of neuroinflammation in the macrosphere model of arterio-arterial embolic focal ischemia: an approximation to human stroke patterns. Exp Transl Stroke Med. 2010 Dec 20;2(1):22. doi: 10.1186/2040-7378-2-22. PMID: 21171972; PMCID: PMC3024233.
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Animal models of post-ischemic forced use rehabilitation: methods, considerations, and limitations.

Experimental & translational stroke medicine

Livingston-Thomas JM, Tasker RA.
PMID: 23343500
Exp Transl Stroke Med. 2013 Jan 23;5(1):2. doi: 10.1186/2040-7378-5-2.

Many survivors of stroke experience arm impairments, which can severely impact their quality of life. Forcing use of the impaired arm appears to improve functional recovery in post-stroke hemiplegic patients, however the mechanisms underlying improved recovery remain unclear. Animal...

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Livingston-Thomas JM, Tasker RA. Animal models of post-ischemic forced use rehabilitation: methods, considerations, and limitations. Exp Transl Stroke Med. 2013;5(1):2doi: 10.1186/2040-7378-5-2.
Livingston-Thomas, J. M., & Tasker, R. A. (2013). Animal models of post-ischemic forced use rehabilitation: methods, considerations, and limitations. Experimental & translational stroke medicine, 5(1), 2. https://doi.org/10.1186/2040-7378-5-2
Livingston-Thomas, Jessica M, and Tasker, R Andrew. "Animal models of post-ischemic forced use rehabilitation: methods, considerations, and limitations." Experimental & translational stroke medicine vol. 5,1 (2013): 2. doi: https://doi.org/10.1186/2040-7378-5-2
Livingston-Thomas JM, Tasker RA. Animal models of post-ischemic forced use rehabilitation: methods, considerations, and limitations. Exp Transl Stroke Med. 2013 Jan 23;5(1):2. doi: 10.1186/2040-7378-5-2. PMID: 23343500; PMCID: PMC3605246.
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Brain edema formation correlates with perfusion deficit during the first six hours after experimental subarachnoid hemorrhage in rats.

Experimental & translational stroke medicine

Westermaier T, Stetter C, Raslan F, Vince GH, Ernestus RI.
PMID: 22551223
Exp Transl Stroke Med. 2012 Jul 13;4(1):8. doi: 10.1186/2040-7378-4-8.

BACKGROUND: Severe brain edema is observed in a number of patients suffering from subarachnoid hemorrhage (SAH). Little is known about its pathogenesis and time-course in the first hours after SAH. This study was performed to investigate the development of...

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Westermaier T, Stetter C, Raslan F, et al. Brain edema formation correlates with perfusion deficit during the first six hours after experimental subarachnoid hemorrhage in rats. Exp Transl Stroke Med. 2012;4(1):8doi: 10.1186/2040-7378-4-8.
Westermaier, T., Stetter, C., Raslan, F., Vince, G. H., & Ernestus, R. I. (2012). Brain edema formation correlates with perfusion deficit during the first six hours after experimental subarachnoid hemorrhage in rats. Experimental & translational stroke medicine, 4(1), 8. https://doi.org/10.1186/2040-7378-4-8
Westermaier, Thomas, et al. "Brain edema formation correlates with perfusion deficit during the first six hours after experimental subarachnoid hemorrhage in rats." Experimental & translational stroke medicine vol. 4,1 (2012): 8. doi: https://doi.org/10.1186/2040-7378-4-8
Westermaier T, Stetter C, Raslan F, Vince GH, Ernestus RI. Brain edema formation correlates with perfusion deficit during the first six hours after experimental subarachnoid hemorrhage in rats. Exp Transl Stroke Med. 2012 Jul 13;4(1):8. doi: 10.1186/2040-7378-4-8. PMID: 22551223; PMCID: PMC3398845.
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Proteomic approach with LCMS-IT-TOF identified an increase of Rab33B after transient focal cerebral ischemia in mice.

Experimental & translational stroke medicine

Hyakkoku K, Hamanaka J, Tsuruma K, Shimazawa M, Hara H.
PMID: 21092243
Exp Transl Stroke Med. 2010 Nov 23;2(1):20. doi: 10.1186/2040-7378-2-20.

BACKGROUND: Several proteins are known to be markedly expressed in the brain during cerebral ischemia; however, the changes in protein profiles within the ischemic brain after an ischemic insult have not been fully elucidated. We studied the changes in...

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Hyakkoku K, Hamanaka J, Tsuruma K, et al. Proteomic approach with LCMS-IT-TOF identified an increase of Rab33B after transient focal cerebral ischemia in mice. Exp Transl Stroke Med. 2010;2(1):20doi: 10.1186/2040-7378-2-20.
Hyakkoku, K., Hamanaka, J., Tsuruma, K., Shimazawa, M., & Hara, H. (2010). Proteomic approach with LCMS-IT-TOF identified an increase of Rab33B after transient focal cerebral ischemia in mice. Experimental & translational stroke medicine, 2(1), 20. https://doi.org/10.1186/2040-7378-2-20
Hyakkoku, Kana, et al. "Proteomic approach with LCMS-IT-TOF identified an increase of Rab33B after transient focal cerebral ischemia in mice." Experimental & translational stroke medicine vol. 2,1 (2010): 20. doi: https://doi.org/10.1186/2040-7378-2-20
Hyakkoku K, Hamanaka J, Tsuruma K, Shimazawa M, Hara H. Proteomic approach with LCMS-IT-TOF identified an increase of Rab33B after transient focal cerebral ischemia in mice. Exp Transl Stroke Med. 2010 Nov 23;2(1):20. doi: 10.1186/2040-7378-2-20. PMID: 21092243; PMCID: PMC3002892.
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Analysis of early phase and subsequent phase III stroke studies of neuroprotectants: outcomes and predictors for success.

Experimental & translational stroke medicine

Minnerup J, Wersching H, Schilling M, Schäbitz WR.
PMID: 24502346
Exp Transl Stroke Med. 2014 Feb 07;6(1):2. doi: 10.1186/2040-7378-6-2.

BACKGROUND: Efficacy of neuroprotective treatments for ischemic stroke was not convincingly demonstrated in clinical phase III trials so far, whereas some preceding early phase studies found neuroprotection to be beneficial. We aimed to determine the frequency with which phase...

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Minnerup J, Wersching H, Schilling M, et al. Analysis of early phase and subsequent phase III stroke studies of neuroprotectants: outcomes and predictors for success. Exp Transl Stroke Med. 2014;6(1):2doi: 10.1186/2040-7378-6-2.
Minnerup, J., Wersching, H., Schilling, M., & Schäbitz, W. R. (2014). Analysis of early phase and subsequent phase III stroke studies of neuroprotectants: outcomes and predictors for success. Experimental & translational stroke medicine, 6(1), 2. https://doi.org/10.1186/2040-7378-6-2
Minnerup, Jens, et al. "Analysis of early phase and subsequent phase III stroke studies of neuroprotectants: outcomes and predictors for success." Experimental & translational stroke medicine vol. 6,1 (2014): 2. doi: https://doi.org/10.1186/2040-7378-6-2
Minnerup J, Wersching H, Schilling M, Schäbitz WR. Analysis of early phase and subsequent phase III stroke studies of neuroprotectants: outcomes and predictors for success. Exp Transl Stroke Med. 2014 Feb 07;6(1):2. doi: 10.1186/2040-7378-6-2. PMID: 24502346; PMCID: PMC3922860.
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Showing 1 to 12 of 109 entries